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Re: Lemon Juice!
Nov 17, 2004
Thanks AA!! I'm thrilled myself and hope it works for others too. It just makes so much sense that we should attack acne from the inside instead of just doing it with topical solutions...

I've added something else to this natural approach against acne.. and that is green tea at night... will look further into that... I hope it doesnt clash with the lemons!! ;)

Take care and all the best!
Re: Lemon Juice!
Nov 24, 2004
AA and all.. hope everything is well with you all and that the lemons are working their way into an acne free life for all of us.

I just wanted to share with y'all that for the last week I have been completely clear!! Not one pimple... sure I still have red marks but I trust that with time they will heal... in the meantime make up is covering them.

Also, I have noticed that my skin isn't oily at all. Before the lemons, I had to use those oil absorb sheets once- twice a day... specially when the heat kicked in around 2 -3 pm.

Well I just wanted to share my clearness with y'all and hope this magnificient recipe will work for all of us.

Today I will be going to the coast for a workshop and it is supposed to be 23- 31 C... I hope the heat/humidity doesn't cause any pimples in me... but I'm taking lemons and green tea... the only thing I forgot was my Aloe Vera.. but I guess I can do without it for 2 nights ;) . Will let y'all know!!

Take care and all the best!
Re: Lemon Juice!
Dec 29, 2004
Here is the current model used to understand how a few subatances affect your metabolism. I am a strong believer in the fatty acid balance theory of acne. Summary/short version at th end:

Citric acid increases the activity of acetyl-CoA carboxylase (ACC), an enzyme transforming acetyl-CoA into
malonyl-CoA 8), which suggests the possibility that the administration of citric acid may promote the
synthesis of malonyl-CoA (Fig. 2). Malonyl-CoA inhibits the uptake of fatty acids into mitochondria
because malonyl-CoA strongly inhibits carnitine-palmitoyltransferase I in mitochondria 8).
On the other hand, pyruvate dehydrogenase complex (PDC) in mitochondria is known to be
inactivated under conditions in which the fatty acid oxidation is promoted and activated when the
oxidation is inhibited 9). Therefore, it is expected that when synthesis of malonyl-CoA is promoted, the
activity of PDC will increase, resulting in that oxidation of pyruvic acid and lactic acid is promoted. In
addition, citric acid is an inhibitor of phosphofructokinase, a regulating enzyme in the glycolytic
pathway (Fig. 2), and therefore inhibits the breakdown of glucose. These two effects of citric acid may
be responsible for the promotion of the breakdown of lactic acid after administration of citric acid.

Hydroxycitric Acid: reported avility to block ATP-citratelyse thus reducing citrate buildup which will inturn inhibit ACC leading to more fat burning as the malyonol-CoA concentration will drop. The result of inibiting ACC is increased Acetyl-Coa

Fibrates: The most common and well-researched negative side effect seems to be renal deposits of acytel-CoA. Acytel-CoA is a necessary product of fat oxidation. As I have discussed in previous articles, it is the balance of acytel-CoA that determines nutrient usage. The body needs acytel-CoA to burn fat in the first place, as acytel-CoA must join with a long chain fatty acid to form an acyal-fatty acid complex that can then be actively transported into the mitochondria by CPT. Unfortunately however, extra actyel-CoA is a prime target for ACC. ACC will transform that excess acytel-CoA into malonyl-CoA, which actually inhibits CPT. To complicate things even further, one of the primary pathways for acytel-CoA metabolism is the lipogenesis pathway. In humans this is not such a problem in terms of fat storage, however when it’s activated it can have negative consequences, particularly for our brains.
Hydroxycitric Acid: reported avility to block ATP-citratelyse thus reducing citrate buildup which will inturn inhibit ACC leading to more fat burning as the malyonol-CoA concentration will drop. The result of inibiting ACC is increased Acetyl-Coa

Green Tea: when there is a large amount of acytel-CoA, the excess gets shuttled down the lipogenesis pathway. Along the way this excess increases the activity of fatty acid synthase (FAS). Increased FAS activity is known to increase appetite primarily by lowering production of MSH and POMC in the Arcuate Nucleus and Lateral Hypothalamus. the result of excess actyel-CoA production—can be countered by an FAS inhibitor. There are several FAS inhibitors available on the market. High doses of hydroxycitrate (10g per day) or green tea extract are two such options. High quality hydroxycitrate is available in bulk at reasonable prices. I personally have used 3g four times per day with much success when it comes to appetite suppression. However, individual response and mileage may vary. Green tea extract is also a potent inhibitor of FAS, and there are numerous green tea extracts available on the market. It is mainly the ECG component that seems to be the strong FAS inhibitor, so I would look for an extract that has a large proportion of ECG.

Short Version: Citric acid disposes Acetyl-Coa, decreases acne. Hydroxycitric acid increases Acetyl-Coa, ??? acne. Fibrates increase Acetyl-CoA, and i think has little effect on acne but not sure. Green tea disposes Acetyl-CoA, decreases acne.

Furthermore, pantothenic acid increases Acetyl-Coa, decreases acne.

It's late, but the Acetyl-CoA levels are less important. It seems like the common bond is the beta-oxidation. Malonyl-CoA is something that pops up in all of these things... i'm just not smart enough to see what it is.

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