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For my diagnosis of SLE in 2005 I had photosensitivity,oral ulcers,arthritis,brain vasculitis or lupus cerebritis,developed anemia later, had a positive anti-DNA , false postive VDRL (syphilis test),and a positive speckled ANA Titre of 1:320. My C4 complement and C3 complement was way below normal. My sed rate was 106. Other labs indicated I was aquiring kidney damage so I was placed on Cellcept right away. Luckily, this reversed the kidney damage.

For the Antiphospholipid syndrome I had: Migraines, brain fog, vertigo and dizziness, visual disturbances including Amaurosis fugax. It is a loss of vision in one eye due to a temporary lack of blood flow to the retina. It was like a gray curtain being pulled down over half of my right eye. I didn't loose full vision. I also had double vision. I had no evidence of stroke. I had many CT scans and MRI's and blood work.

My positive bloodwork for the diagnosis that was all positive:

Anticardiolipin antibodies (IgG, IgM, and IgA)

Lupus anticoagulant a panel of blood clotting tests that may include the dilute Russel Viper venom time (dRVVT), lupus aPTT, mixing studies, and hex phase phospholipid test, platelet neutralization procedure

Antibodies to b2-glycoprotein I (IgG, IgM, IgA)

I see both a Rheumatologist and a Hematologist.

The warm antibodies are a type of autoimmune hemolytic anemia.In some types of AIHA, the antibodies made by the body are called warm antibodies. This means they're active (that is, they destroy red blood cells) at warm temperatures, such as body temperature. Some conditions are worse then others.

In other types of AIHA, the body makes cold-reactive antibodies, which become active in colder temperatures. Cold-reactive antibodies can become active when parts of the body, such as the hands or feet, are exposed to temperatures lower than 32 to 50 degrees.

Warm antibody AIHA is more common than cold-reactive antibody AIHA.

This makes is very hard for me to have a blood transfusion, given these antibodies as well as I am RH Negative.

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